Background: Axonal degeneration by the Nogo receptor-1 (NgR1) signaling was recently recognized
as an important factor in pathogenesis of multiple sclerosis (MS). We previously identified
lateral olfactory tract usher substance (LOTUS) as an endogenous NgR1 antagonist and
showed that the reduction of LOTUS in the cerebrospinal fluid correlated with the
disease activity of MS.
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