« Previous
Next »
Journal of the Neurological Sciences
Volume 222, Issue 1
, Pages 65-73
, 15 July 2004
Time-dependent cytokine deviation toward the Th2 side in Japanese multiple sclerosis patients with interferon beta-1b
References
- . The epigenetics of multiple sclerosis: clues to etiology and a rationale for immune therapy. Annu. Rev. Neurosci. 1994;17:247–265
- . Immunopathogenesis of multiple sclerosis: the role of T cells. Curr. Opin. Neurol. 1999;12:309–321
- . Interferon beta in the treatment of multiple sclerosis: mechanisms of action. Neurology. 1998;51:682–689
- . Interferon-gamma-secreting cells in multiple sclerosis patients treated with interferon beta-1b. Neurology. 1995;45:2173–2177
- . Incidence of exacerbations in the first 90 days of treatment with recombinant human interferon beta-1b in patients with relapsing-remitting multiple sclerosis. Ann. Neurol. 1998;44:138–139
- Interferon-β treatment in multiple sclerosis patients decreases the number of circulating T cells producing interferon-gamma and interleukin-4. J. Neuroimmunol. 2000;111:86–92
- . Interferon-β in multiple sclerosis: altering the balance of interleukin-12 and interleukin-10?. Curr. Opin. Neurol. 2001;14:361–368
- Western versus Asian types of multiple sclerosis: immunogenetically and clinically distinct disorders. Ann. Neurol. 1996;40:569–574
- HLA-DPB1*0501-associated opticospinal multiple sclerosis: clinical, neuroimaging and immunogenetic studies. Brain. 1999;122:1689–1696
- Recommended diagnostic criteria for multiple sclerosis: guidelines from the International Panel on the diagnosis of multiple sclerosis. Ann. Neurol. 2001;50:121–127
- . Rating neurologic impairment in multiple sclerosis: an expanded disability status scale (EDSS). Neurology. 1983;33:1444–1452
- Tc1/Tc2 and Th1/Th2 balance in Asian and Western types of multiple sclerosis, HTLV-I-associated myelopathy/tropical spastic paraparesis and hyperIgEaemic myelitis. J. Neuroimmunol. 2001;119:297–305
- Complex immunomodulatory effects of interferon-β in multiple sclerosis include the upregulation of T helper 1-associated marker genes. Ann. Neurol. 2001;50:349–357
- Systemic IFN-β treatment induces apoptosis of peripheral immune cells in MS patients. J. Neuroimmunol. 2003;137:187–196
- . Abnormal levels of interferon-gamma receptors in active multiple sclerosis are normalized by IFN-beta therapy: implications for control of apoptosis. Front. Biosci. 2004;9:1547–1555
- . Cytotoxic T lymphocytes in autoimmune and degenerative CNS diseases. Trends Neurosci. 2002;25:313–319
- Increased IL-13 but not IL-5 production by CD4-positive T cells and CD8-positive T cells in multiple sclerosis during relapse phase. J. Neurol. Sci. 2002;201:45–51
- Late complications of immune deviation therapy in a nonhuman primate. Science. 1996;274:2054–2057
- Myelin basic protein-specific T helper 2 (Th2) cells cause experimental autoimmune encephalomyelitis in immunodeficient hosts rather than protect them from the disease. J. Exp. Med. 1997;186:307–312
- . Molecular and biological characteristics of interleukin-13. Chem. Immunol. 1996;63:204–218
- NK cells modulate the cytotoxic activity generated by Mycobacterium leprae-hsp65 in leprosy patients: role of IL-18 and IL-13. Clin. Exp. Immunol. 2004;135:105–113
- . Regulation of experimental autoimmune encephalomyelitis by natural killer (NK) cells. J. Exp. Med. 1997;186:1677–1687
- . The role of NK cells in autoimmune disease. Autoimmunity. 2002;35:1–14
- . Clinical relapses of multiple sclerosis are associated with ‘novel’ valleys in natural killer cell functional activity. J. Neuroimmunol. 2003;145:103–114
- . Interleukin 13, an interleukin 4-like cytokine that acts on monocytes and B cells, but not on T cells. Immunol. Today. 1994;15:19–26
- . Macrophage-inactivating IL-13 suppresses experimental autoimmune encephalomyelitis in rats. J. Immunol. 1994;153:4258–4267
- . IL-4, IL-10 and IL-13 modulate A beta(1–42)-induced cytokine and chemokine production in primary murine microglia and a human monocyte cell line. J. Neuroimmunol. 2001;113:49–62
- Regulation of endothelial and mesothelial cell function by interleukin-13: selective induction of vascular cell adhesion molecule-1 and amplification of interleukin-6 production. Blood. 1994;84:1913–1921
- . IL-13 selectively induces vascular cell adhesion molecule-1 expression in human endothelial cells. J. Immunol. 1995;154:799–803
- . Apoptosis of myelin-reactive T cells induced by reactive oxygen and nitrogen intermediates in vitro. Cell. Immunol. 1997;178:1–8
- . The multiplex function of nitric oxide in (auto)immunity. J. Exp. Med. 1998;187:1361–1365
- . IFN-γ is critical to the control of murine autoimmune encephalomyelitis and regulates both in the periphery and in the target tissue: a possible role for nitric oxide. J. Immunol. 1999;163:5278–5286
- . Macrophage-derived nitric oxide inhibits the proliferation of activated T helper cells and is induced during antigenic stimulation of resting T cells. Cell. Immunol. 2000;199:43–49
- A comparative study of Japanese multiple sclerosis patients with and without oligoclonal IgG bands. Mult. Scler. 2002;8:459–462
- Th1 dominance in HAM/TSP and the optico-spinal form of multiple sclerosis versus Th2 dominance in mite antigen-specific IgE myelitis. J. Neurol. Sci. 2000;172:17–24
- Flow cytometric differentiation of Asian and Western types of multiple sclerosis, HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and hyperIgEaemic myelitis by analyses of memory CD4 positive T cell subsets and NK cell subsets. J. Neurol. Sci. 2000;177:24–31
PII: S0022-510X(04)00108-X
doi: 10.1016/j.jns.2004.04.012
© 2004 Elsevier B.V. All rights reserved.
« Previous
Next »
Journal of the Neurological Sciences
Volume 222, Issue 1
, Pages 65-73
, 15 July 2004
